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A Clinician's Guide to Treatment

References 1. Bissada H. et al.: Olanzapine in the treatment of low body weight and obsessive thinking in women with anorexia nervosa: a randomized, double-blind, placebo-controlled trial. Am J Psychiatry, 165, 10,2008. 2. Court A. et al.: Investigating the effectiveness, safety and tolerability of quetiapine in the treatment of anorexia nervosa in young people: a pilot study. J Psychiatr Res., 44, 15, 2010. 3. Hagman J. et al.: A double-blind, placebo-controlled study of risperidone for the treatment of adolescents and young adults with anorexia nervosa: a pilot

Introduction The hallmark of AN is a significantly reduced body weight associated with mortality 12 times higher than all other causes of death in adolescence and early adulthood in females [ 1 ]. The Diagnostic and Statistical Manual of Mental Disorders, 5th ed. (DSM-V) [ 2 ] criteria for anorexia nervosa (AN) include in short: (A) restricted energy intake, so that a significantly low body weight ensues, (B) intense fear to gain weight, and (C) severely disturbed body weight or shape experience. There are also defined subtypes: (1) restricting type (weight loss

was considered by the team at the hospital, anorexia nervosa was in the differential to explain the patient’s symptoms of anorexia, abdominal pain, and weight loss. The Diagnostic and Statistical Manual of Mental Disorders (DSM) IV criteria for anorexia nervosa requires a patient to have a manifest refusal to maintain weight that is appropriate for one’s age (with 85% of ideal body weight as the cut off); a fear of gaining weight or becoming fat, despite having low body weight; a disturbance in the way one’s body weight or shape is experienced; undue influence of

DOI 10.1515/jpem-2012-0193      J Pediatr Endocr Met 2013; 26(1-2): 167–172 Patient report Jennifer Galle * , Susan Kirsch and Miriam Kaufman Anorexia nervosa in a patient with congenital adrenal hyperplasia Abstract: Life stressors and hormonal dysregulation in the hypothalamic-pituitary-adrenal (HPA) axis are common factors that play a role in both anorexia nervosa (AN) and congenital adrenal hyperplasia (CAH). The com- plexity of combined CAH and AN on the HPA axis has not, to our knowledge, been previously illustrated. We present a report

REFERENCES 1. American Psychiatric Association. Diagnostic and statistical manual of mental disorders, 5th ed. Washington, DC: APA; 2013. 2. Keating C. Theoretical perspective on anorexia nervosa: The conflict of reward. Neurosci Biobehav Rev. 2010;34:73-9. 3. Birmingham CL, Treasure J. Medical management of eating disorders 2nd edition. Cambridge: Cambridge University Press; 2011. 4. Jáuregui-Garrido B, Jáuregui-Lobera I. Sudden death in eating disorders. Vasc Health Risk Manag. 2012;8:91-8. 5. NICE. Eating disorders: core interventions in the treatment and

[ 8 – 12 ], VASP is suggested to be a visceral adipose-derived adipokine with insulin-sensitizing effects [ 13 ]. It has recently been suggested that VASP may also play a role in the regulation of food intake and modulation of eating behavior [ 14 – 16 ]. Anorexia nervosa (AN) is a chronic psychosomatic syndrome that is characterized by abnormal eating behavior. The essence of AN is the person striving to obtain a slim silhouette by deliberately limiting the amount of food eaten, undergoing strenuous physical exercise and inducing vomiting or using laxatives and

Introduction Anorexia nervosa is a medical and psychological disorder that affects 0.5% of adolescent girls in the USA (1). It is classically seen in young women who present with significant weight loss, a distorted body image, and an intense fear of gaining weight despite being underweight. The diagnosis is a clinical one and is usually made after other organic pathologies have been ruled out. Patients have often been evaluated by multiple subspecialists before being referred for an eating disorder evaluation. A much more rare diagnosis that is also associated

References 1. Boghi A., Sterpone S., Sales S., D'Agata F., Bradac G.B., Zullo G., et al. In vivo evidence of global and focal brain alterations in ano-rexia nervosa. Psychiatry Res., 2011; 192: 154–159; DOI: 10.1016/j.pscychresns.2010.12.008. 2. Muhlau M., Gaser C., Ilg R., Conrad B., Leibl C., Cebulla M.H., et al. Gray matter decrease of the anterior cingulate cortex in anorexia nervosa. Am. J. Psychiatr., 2007; 64: 1850–1857; DOI: 10.1176/appi.ajp.2007.06111861. 3. Swayze VW., Andersen AE., Andreasen NC., Arndt S., Sato Y., Ziebell S. Brain tissue volume

Introduction Constitutional thinness has been defined as having an extremely low body weight (low body mass index [BMI]), while having close-to-normal body composition (normal amounts of fat tissue), normal menstrual cycles, normal hormonal profile and equilibrated or positive energy metabolism in the absence of anorexia nervosa (AN) diagnostic criteria [ 1 ], [ 2 ], [ 3 ]. Adolescence is an essential period for bone mineralization, as the acquisition of approximately 40–60% of adult bone mass takes place during this period of life [ 4 ]. The negative impact of