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References American College of Rheumatology Task Force on Osteoporosis Guidelines. Recommendations for the prevention and treatment of glucocorticoid-induced osteoporosis. Arthritis Rheum 1996;39:1791-801. Mazziotti G, Angeli A, Bilezikian JP, Canalis E, Giustina A. Glucocortcoid-induced osteoporosis: an update. Trends Endocrnol Metab 2006;17:144-9. Lukert BP, Raisz LG. Glucocorticoid-induced osteoporosis: pathogenesis and management. Ann Intern Med 1990:112:352-64. Shaker JL, Lukert BP. Osteoporosis associated with excess glucocorticoids. Endocrinol Metab

Mammalia 74 (2010): 347–350 2010 by Walter de Gruyter • Berlin • New York. DOI 10.1515/MAMM.2010.032 2010/023 Article in press - uncorrected proof Short Note Seasonal glucocorticoid secretion in mountain hares (Lepus timidus) Maik Rehnus1,*, Rupert Palme2, Flurin Filli3 and Klaus Hackländer1 1 Institute of Wildlife Biology and Game Management, University of Natural Resources and Life Sciences Vienna, Gregor-Mendel-Str. 33, 1180 Vienna, Austria, e-mail: maik.rehnus@gmx.de 2 Department of Biomedical Sciences/Biochemistry, University of Veterinary Medicine Vienna

BioMol Concepts, Vol. 3 (2012), pp. 495–504 • Copyright © by Walter de Gruyter • Berlin • Boston. DOI 10.1515/bmc-2012-0012 Review The infl uence of glucocorticoids on neuronal survival and synaptic function Tadahiro Numakawa 1,2, *, Naoki Adachi 1,2 , Misty Richards 1,3 , Shuichi Chiba 1,4 and Hiroshi Kunugi 1,2 1 Department of Mental Disorder Research , National Institute of Neuroscience, National Center of Neurology and Psychiatry, Tokyo 187-8502 , Japan 2 Core Research for Evolutional Science and Technology Program (CREST

Horm Mol Biol Clin Invest 2010;4(2):559–564 2010 by Walter de Gruyter • Berlin • New York. DOI 10.1515/HMBCI.2010.051 2010/025 Article in press - uncorrected proof Glucocorticoid signaling in cardiac disease Rongqin Ren and John A. Cidlowski* Laboratory of Signal Transduction, Molecular Endocrinology Group, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, NC 27709, USA Abstract As major mediators of stress regulation, glucocorticoids have an essential role in maintaining cardiovascular homeostasis under

Introduction Secretion of glucocorticoids (GC) by the adrenal cortex is under control of a neuroendocrine feedback system, the hypothalamo-pituitary-adrenal (HPA) axis. Initiated by the secretion of hypothalamic corticotropin releasing hormone (CRH), pituitary pro-opiomelanocortin (POMC) gene transcription, and secretion of the POMC-encoded adrenocorticotropic hormone (ACTH) are induced, the latter of which stimulates adrenal GC synthesis and secretion. This activation cascade is inhibited by the GR at the level of the hypothalamus and the anterior pituitary [1

Introduction Fifteen million infants are born prematurely each year, a number that is expected to continue to increase [ 1 ]. Additionally, the use of antenatal glucocorticoids in women with preterm labor has dramatically improved outcomes for premature infants [ 2 ]. Glucocorticoids (commonly betamethasone and dexamethasone) can be administered as an intramuscular injection and have effects lasting about a week. Antenatal use of these medications is associated with a reduced risk of intraventricular hemorrhage, respiratory distress syndrome (RDS) and perinatal

GLUCOCORTICOID RECEPTORS Mohammed Kalimi and John R. Hubbard Department of Physiology and Biophysics Medical College of Virginia Virginia Commonwealth University Richmond, Virginia 23298 U.S.A. I. Introduction II. Measurement of the glucocorticoid receptor III. Relationship between receptor binding and biological activity IV. Clinical applications of the receptor assay V. Hormone agonist/antagonist selection by receptor analysis VI. Glucocorticoid receptor microheterogeneity and separation by chromatographic techniques VII. Receptor modulation in

J Pediatr Endocr Met 2012;25(3-4):307–312 © 2012 by Walter de Gruyter • Berlin • Boston. DOI 10.1515/jpem-2011-0441 *Corresponding author: Pornpimol Rianthavorn, MD, Division of Nephrology, Department of Pediatrics, 1873 King Chulalongkorn Memorial Hospital, Bangkok 10330, Thailand Phone: + 66-2-256-4951, Fax: + 66-2-256-4911, E-mail: Pornpimol.r@chula.ac.th Received October 27, 2011; accepted January 9, 2012; previously published online February 27, 2012 Prevention of bone loss in children receiving long-term glucocorticoids with calcium and

Introduction The glucocorticoid hormones, especially the synthetic analogs, such as prednisone and dexamethasone, are the most potent known anti-inflammatory drugs [1]. However, their usefulness is limited by the high risk of the serious side effects such as osteoporosis and insulin resistance [1–3]. Over the past 50 years or so, there has been a search for the analogs of the glucocorticoids that would bind to the glucocorticoid receptors responsible for the anti-inflammatory effects with minimal adverse effects. To date, such a search has been unrewarding

DOI 10.1515/revneuro-2012-0058   Rev. Neurosci. 2012; 23(5-6): 681–690 Research Article Daniela Hauer* , Florian Weis , Patrizia Campolongo , Mirjam Schopp , Andres Beiras- Fernandez , Claudia Strewe , Markus Giehl , Roland Toth , Erich Kilger and Gustav Schelling Glucocorticoid-endocannabinoid interaction in cardiac surgical patients: relationship to early cognitive dysfunction and late depression Abstract Background: Endocannabinoids (ECs) are rapidly acting immune-modulatory lipid-signaling molecules that are important for