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References 1 Bence Jones H. Papers on chemical pathology – lecture III. Lancet 1847; 2:88–92. 2 Whicher JT, Banks RE. Cytokines as tumour markers. Scand J Clin Lab Invest 1995; 55:122–44. 3 Dunlop RJ, Campbell CW. Cytokines and advanced cancer. J Pain Symptom Manage 2000; 20:214–32. 4 Cohen S, Bigazzi PE, Yoshida T. Similarities of T cell function in cell-mediated immunity and antibody production. Cell Immunol 1974; 12:150–9. 5 Berdel WE, Danhauser RS, Oberberg D, Zafferani M. Effects of hematopoietic growth factors on malignant nonhematopoietic cells. Semin

and intraepithelial lymphocytes . Gastroenterology. 1989; 97 (6):1562-84. 4. GRISHAM MB, YAMADA T. Neutrophils, nitrogen oxides, and inflammatory bowel disease. Annals of the New York Academy of Sciences. 1992; 664 :103-15. 5. FIOCCHI CB, DG. KATZ, JA. Cytokine production in the human gastrointestinal tract during inflammation . Current Opinion in Gastroenterology. 1994; 10 (6):639-44. 6. RAGHOW R. T he role of extracellular matrix in postinflammatory wound healing and fibrosis . FASEB journal: official publication of the Federation of American Societies for

Seki et al.: Regulation of neopterin production b\' cytokincs Pteridines Vol. 7, 1996, pp. 5 - 9 The Regulation of Neopterin Production by Cytokines Takashi Seki l §, Nobuyuki Sugie\ Kosuke Johl, Tsutomu Oh-ishil 1 Division of Infectious Diseases, Immunology and Allergy, Saitama Children's Medical Center, Iwatsuki, Saitama, Japan. 2 Department of Pediatrics, National Kamaishi Hospital, Kamaishi, Iwate, Japan . (Received January J6, 1996) Summary While cytokines are produced by various cells; T cells, B cells, NK cells, monocytes, vascular en- dothelial

mediators and stimulate production of pain signaling molecules (e.g., glutamate, substance P, calcitonin gene-related peptide); prolonged release of pro-inflammatory mediators can cause central nervous system changes that may result in neuropathic pain [ 9 ]. As various shared mechanisms are identified between the two types of pain, they warrant reconsideration of our understanding, diagnosis, and treatment of both neuropathic and inflammatory pain [ 10 ]. The signaling molecules of the immune system are cytokines, which can be broadly categorized as either pro

J. Perinat. Med. 39 (2011) 731–736 • Copyright by Walter de Gruyter • Berlin • Boston. DOI 10.1515/JPM.2011.078 2011/0007 Article in press - uncorrected proof NICU admission hypothermia, chorioamnionitis, and cytokines Karen D. Fairchild1,*, Chen-Chih J. Sun2, George C. Gross3, Adora C. Okogbule-Wonodi4, Rose M. Chasm4 and Rose M. Viscardi4 1 Department of Pediatrics, University of Virginia School of Medicine, Charlottesville, VA, USA 2 Department of Pathology, University of Maryland School of Medicine, Baltimore, MD, USA 3 Department of Radiology, University of

upregulate iNOS and NO production and inhibit chlamydial growth. PLoS One, 2012, 7: e39214. 10 Yilma AN, Singh SR, Fairley SJ, et al. The anti-inflammatory cytokine, interleukin-10, inhibits inflammatory mediators in human epithelial cells and mouse macrophages exposed to live and UV-inactivated Chlamydia trachomatis. Mediators Inflamm, 2012, DI 2012:520174. 11 Wang C, Tang J, Geisler WM, et al . Human leukocyte antigen and cytokine gene variants as predictors of recurrent Chlamydia trachomatis infection in high-risk adolescents. J Infect Dis, 2005, 191:1084-1092. 12

References 1 Tobita K, Kijima H, Dowaki S, Kashiwagi H, Ohtani Y, Oida Y, et al. Epidermal growth factor receptor expression in human pancreatic cancer: significance for liver metastasis. Int J Mol Med 2003; 11: 305–9. 2 Bardeesy N, DePinho RA. Pancreatic cancer biology and genetic. Cancer 2002; 2: 897–909. 3 Uematsu T, Tsuchie K, Ukai K, Kimoto E, Funakawa T, Mizuno R. Granulocyte-colony stimulating factor produced by pancreatic carcinoma. Int J Pancr 1996; 9: 135–9. 4 Dunlop RJ, Campbell CW. Cytokines and advanced cancer. J Pain Sympt Manage 2000; 20: 214

J Pediatr Endocr Met 2011;24(7-8):405–410 © 2011 by Walter de Gruyter • Berlin • Boston. DOI 10.1515/JPEM.2011.243 Review Bone disease and cytokines in idiopathic hypercalciuria: a review Augusto C.S. Santos Jr., Eleonora M. Lima, Eduardo Ara ú jo Oliveira and Ana Cristina Sim õ es e Silva * Department of Pediatrics , Pediatric Nephrology Unit, Hospital das Cl í nicas, Federal University of Minas Gerais, Belo Horizonte , Brazil Abstract Bone remodeling is a continuous and dynamic process of skeletal destruction and renewal. A complex

References 1. Wu GF, Alvarez E. The immunopathophysiology of multiple sclerosis. Neurol Clin 2011;29:257-78. 2. Hollifield RD, Harbige LS, Pham-Dinh D, et al. Evidence for cytokine dysregulation in multiple sclerosis: peripheral blood mononuclear cell production of pro-inflammatory and anti-inflammatory cytokines during relapse and remission. Autoimmunity 2003;36(3):133-41. 3. Brandão CO, Ruocco HH, Farias AS, et al. Cytokines and intrathecal IgG synthesis in multiple sclerosis patients during clinical remission. Arq Neuropsiquiatr 2005;63(4):914-19. 4. Hellings

REFERENCES 1. Stanilova S. Cytokine Gene Polymorphisms in Colorectal Cancer. In: Ettarh R, ed. Colorectal Cancer Biology - From Genes to Tumor. 1st ed. Rijeka: InTech; 2012:59-78. 2. Marin I, Kipnis J. Learning and memory ... and the immune system. Learn Mem 2013;20(10):601-6. 3. McAfoose J, Baune BT. Evidence for a cytokine model of cognitive function. Neurosci Biobehav Rev 2009;33:355-366. 4. Coogan AN, O’Neill LA, O’Connor JJ. The P38 mitogen-activated protein kinase inhibitor SB203580 antagonizes the inhibitory effects of interleukin-1beta on long