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References Ozsoy R, Van der Steg W, Kastelein J, Arisz L, Koopman M. Dyslipidaemia as predictor of progressive renal failure and the impact of treatment with atorvastatin. Nephrol Dial Transplant 2007; 22: 1578-86. Ozsoy RC, Kastelein JJP, Arisz L, Koopman MG. Atorvastatin and the dyslipidemia of early renal failure. Atherosclerosis 2003; 166: 187-94. Himmelfarb J, Stenvinkel P, Ikizler TA, Hakim RM. The elephant in uremia: oxidant stress as a unifying concept of cardiovascular disease in uremia. Kidney Int 2002; 62: 1524-38. McCullough PA. Why is chronic

, dialysis, chronic renal insufficiency. Available at: http://web.emmes.com/ study/ped/annlrept/annlrept 2006.pdf 18. De Graaff LC, Mulder PG, Hokken-Koelega AC. Body proportions before and during growth hormone therapy in children with chronic renal failure. Pediatr Nephrol. 2003; 18:679-68. 19. Qayyum N, Alcocer L, Maxwell H, Beattie TJ, Murphy AV, Ramage IJ, Ahmed SF. Skeletal disproportion in children with chronic renal disease. Horm Res. 2003; 60:221-226. 20. Abitbol CL, Warady BA, Massie MD, Baluarte HJ, Fleischman LE, Geary DF, Kaiser BA, McEnery PT, Chan JC. Linear

Introduction Renal failure is characterized by a loss of kidney function which may be transient or irreversible, but constantly presents a typical biological feature, hyperuremia, which increases the intensity of a non-enzymatic post-translational modification of proteins, carbamylation. Carbamylation belongs to a group of chemical reactions which alter structural and functional properties of proteins and lead to the formation of numerous byproducts called post-translational modifications derived products (PTMDPs) [1]. This peculiar reaction is characterized by

DOI 10.1515/jpem-2012-0176   J Pediatr Endocr Met 2012; 25(9-10): 1027–1030 Ah Reum Kwon , Jung-Min Ann , Jae Il Shin , Hyun Wook Chae and Ho-Seong Kim* Hypodipsic hypernatremia leading to reversible renal failure following surgery for craniopharyngioma Abstract: Thirst is stimulated by increases in effective plasma osmolality that are detected by cerebral osmore- ceptors located in the vascular organ of the lamina ter- minalis. However, surgical destruction or organic lesions of the lamina terminalis decrease the sensation of thirst in

References Joshi B, Jones D, Rochford A, Giblin L. Hypothyroidism and associated acute renal failure. J R Soc Med 102, 199–200, 2009. Little WC, Freeman GL. Pericardial disease. Circulation 113, 1622–1632, 2006. Patil VC, Patil HV, Agrawal V, Patil S. Cardiac tamponade in a patient with primary hypothyroidism. Indian J Endocrinol Metab 15, S144–S146, 2011. Rodrigo C, Gamakaranage CS, Epa DS, Gnanathasan A, Rajapakse S. Hypothyroidism causing paralytic ileus and acute kidney injury – case report. Thyroid Res 4, 7, 2011. Sarnak MJ, Levey AS, Schoolwerth AC, Coresh

infarction, N. Engl. J. Med., 2004, 351, 1285–1295 http://dx.doi.org/10.1056/NEJMoa041365 [12] Amann K., Tyralla K., Cardiovascular changes in chronic renal failure — pathogenesis and therapy, Clinical Nephrol., 2002, 58, S62–S72 [13] Dries D.L., Exner D.V., Domanski M.J., The prognostic implications of renal insufficiency in asymptomatic and symptomatic patients with left ventricular systolic dysfunction, J. Am. Coll. Cardiol., 2000, 35, 681–689 http://dx.doi.org/10.1016/S0735-1097(99)00608-7 [14] Go A.S., Chertow G.M., Fan D., Chronic kidney disease and the risks of

Drug Metab Drug Interact 2011;26(2):79–80 © 2011 by Walter de Gruyter • Berlin • Boston. DOI 10.1515/DMDI.2011.106 Rhabdomyolysis-induced acute renal failure due to itraconazole and simvastatin association S é bastien Roques, Maria Lytrivi, Daniel Rusu, Jacques Devriendt and David De Bels * Intensive Care Department , Brugmann University Hospital, Brussels , Belgium Abstract We present the case of an 82-year-old man admitted to our hospital for muscle weakness. He was under simvastatin 20 mg per day and was given pulse itraconazole

References 1. Pang, T., Bhutta, Z.A., Finlay, B.B. & Altwegg, M. (1995). Typhoid fever and other salmonellosis: a continuing challenge. Trends Microbiol. 3(7), 253-255. 2. Khan, M., Coovadia, Y. & Sturm, A.W. (1998). Typhoid fever complicated by acute renal failure and hepatitis: case reports and review. Am J Gastroenterol. 93(6), 1001-1003. doi: 10.1111/j.1572-0241.1998.298_y.x 3. Khosla, S.N., Singh, R., Singh, G.P. & Trehan, V.K. (1988). The spectrum of hepatic injury in enteric fever. Am J Gastroenterol. 83(4), 413-416. 4. Pramoolsinsap, C. & Viranuvatti, V

[1] Adaszek L., Winiarczyk S. 2008. Molecular characterization of Babesia canis canis isolates from naturally infected dogs in Poland. Veterinary Parasitology, 152, 235–241. DOI: 10.1016/j.vetpar.2007.12.024. http://dx.doi.org/10.1016/j.vetpar.2007.12.024 [2] Albright R.C. Jr. 2001. Acute renal failure: a practical update. Mayo Clinic Proceedings, 76, 67–74. DOI: 10.1016/S0025-6196(11)62082-X. http://dx.doi.org/10.4065/76.1.67 [3] Cowgill L.D., Langston C. 2011. Acute kidney insufficiency. In: (Eds. Bartges J., Polzin D.J.) Nephrology and Urology of Small Animals

Introduction The study of establishment of a chronic renal failure (CRF) model has been made in many animals including guinea pigs, rabbits, cats, sheep, dogs, rats, and mice ( 6 , 20 ). A CRF model can be established through physical, chemical or biological methods, but animals with renal tissue resection have been held up as a successful CRF model ( 8 ). Alevy et al . ( 1 ) performed renal artery ligation and removal of the contralateral kidney attempting to establish a rat CRF model. However, it was difficult to operate on the animal and easy to cause its