[1] Harukuni I., Bhardwaj A., Mechanisms of brain injury after global cerebral ischemia, Neurol. Clin., 2006, 24, 1–21 http://dx.doi.org/10.1016/j.ncl.2005.10.004CrossrefGoogle Scholar
[2] Sandoval K.E., Witt K.A., Blood-brain barrier tight junction permeability and ischemic stroke, Neurobiol. Dis., 2008, 32, 200–219 http://dx.doi.org/10.1016/j.nbd.2008.08.005Web of ScienceCrossrefGoogle Scholar
[3] LeBleu V.S., Macdonald B., Kalluri R., Structure and function of basement membranes, Exp. Biol. Med. (Maywood), 2007, 232, 1121–1129 http://dx.doi.org/10.3181/0703-MR-72Web of ScienceCrossrefGoogle Scholar
[4] Heo J.H., Lucero J., Abumiya T., Koziol J.A., Copeland B.R., et al., (1999) Matrix metalloproteinases increase very early during experimental focal cerebral ischemia, J. Cereb. Blood Flow Metab., 1999, 19, 624–633 http://dx.doi.org/10.1097/00004647-199906000-00005CrossrefGoogle Scholar
[5] Wang X., Mori T., Jung J.C., Fini M.E., Lo E.H., Secretion of matrix metalloproteinase-2 and -9 after mechanical trauma injury in rat cortical cultures and involvement of MAP kinase, J. Neurotrauma, 2002, 19, 615–625 http://dx.doi.org/10.1089/089771502753754082CrossrefGoogle Scholar
[6] Rosenberg G.A., Navratil M., Barone F., Feuerstein G., Proteolytic cascade enzymes increase in focal cerebral ischemia in rat, J. Cereb. Blood Flow Metab., 1996, 16, 360–366 http://dx.doi.org/10.1097/00004647-199605000-00002CrossrefGoogle Scholar
[7] Hashimoto T., Wen G., Lawton M.T., Boudreau N.J., Bollen A.W., et al., Abnormal expression of matrix metalloproteinases and tissue inhibitors of metalloproteinases in brain arteriovenous malformations, Stroke, 2003, 34, 925–931 http://dx.doi.org/10.1161/01.STR.0000061888.71524.DFCrossrefGoogle Scholar
[8] Zhang X., Pan X.L., Liu X.T., Wang S., Wang L.J., Down-regulation of platelet-activating factor receptor gene expression during focal reversible cerebral ischemia in rats, Neurochem. Res., 2007, 32, 451–456 http://dx.doi.org/10.1007/s11064-006-9248-yCrossrefGoogle Scholar
[9] Pfefferkorn T., Rosenberg G.A., Closure of the blood-brain barrier by matrix metalloproteinase inhibition reduces rtPA-mediated mortality in cerebral ischemia with delayed reperfusion, Stroke, 2003, 34, 2025–2030 http://dx.doi.org/10.1161/01.STR.0000083051.93319.28CrossrefGoogle Scholar
[10] Sumii T., Lo E.H., Involvement of matrix metalloproteinase in thrombolysis-associated hemorrhagic transformation after embolic focal ischemia in rats, Stroke, 2002, 33, 831–836 http://dx.doi.org/10.1161/hs0302.104542CrossrefGoogle Scholar
[11] Higdon J.V., Frei B., Tea catechins and polyphenols: health effects, metabolism., and antioxidant functions, Crit. Rev. Food Sci. Nutr., 2003, 43, 89–143 http://dx.doi.org/10.1080/10408690390826464CrossrefGoogle Scholar
[12] Muia C., Mazzon E., Di Paola R., Genovese T., Menegazzi M., et al., Green tea polyphenol extract attenuates ischemia/reperfusion injury of the gut, Naunyn Schmiedebergs Arch. Pharmacol., 2005, 371, 364–374 http://dx.doi.org/10.1007/s00210-005-1076-0CrossrefGoogle Scholar
[13] Lee S.Y., Kim C.Y., Lee J.J., Jung J.G., Lee S.R., Effects of delayed administration of (-)-epigallocatechin gallate, a green tea polyphenol on the changes in polyamine levels and neuronal damage after transient forebrain ischemia in gerbils, Brain Res. Bull., 2003, 61, 399–406 http://dx.doi.org/10.1016/S0361-9230(03)00139-4CrossrefGoogle Scholar
[14] Aneja R., Hake P.W., Burroughs T.J., Denenberg A.G., Wong H.R., et al., Epigallocatechin, a green tea polyphenol, attenuates myocardial ischemia reperfusion injury in rats, Mol. Med., 2004, 10, 55–62 http://dx.doi.org/10.2119/2004-00032.AnejaCrossrefGoogle Scholar
[15] Yamaguchi M., Calvert J.W., Kusaka G., Zhang J.H., One-stage anterior approach for four-vessel occlusion in rat, Stroke, 2005, 36, 2212–2214 http://dx.doi.org/10.1161/01.STR.0000182238.08510.c5CrossrefGoogle Scholar
[16] Ostrowski R.P., Colohan A.R., Zhang J.H., Mechanisms of hyperbaric oxygen-induced neuroprotection in a rat model of subarachnoid hemorrhage, J. Cereb. Blood Flow Metab., 2005, 25, 554–571 http://dx.doi.org/10.1038/sj.jcbfm.9600048CrossrefGoogle Scholar
[17] Yang C.S., Ju J., Lu G., Xiao H., Hao X., et al., Cancer prevention by tea and tea polyphenols, Asia Pac. J. Clin. Nutr. 17(Suppl. 1), 245–248 Google Scholar
[18] Sabu M.C., Smitha K., Kuttan R., Anti-diabetic activity of green tea polyphenols and their role in reducing oxidative stress in experimental diabetes, J. Ethnopharmacol., 2002, 83, 109–116 http://dx.doi.org/10.1016/S0378-8741(02)00217-9CrossrefGoogle Scholar
[19] Cavet M.E., Harrington K.L., Vollmer T.R., Ward K.W., Zhang J.Z., Antiinflammatory and anti-oxidative effects of the green tea polyphenol epigallocatechin gallate in human corneal epithelial cells, Mol. Vis., 2011, 17, 533–542 Google Scholar
[20] Bhardwaj A., Alkayed N.J., Kirsch J.R., Hurn P.D., Mechanisms of ischemic brain damage, Curr. Cardiol. Rep., 2003, 5, 160–167 http://dx.doi.org/10.1007/s11886-003-0085-1CrossrefGoogle Scholar
[21] Kahle K.T., Simard J.M., Staley K.J., Nahed B.V., Jones P.S., et al., Molecular mechanisms of ischemic cerebral edema: role of electroneutral ion transport, Physiology (Bethesda), 2009, 24, 257–265 http://dx.doi.org/10.1152/physiol.00015.2009CrossrefGoogle Scholar
[22] Unterberg A.W., Stover J., Kress B., Kiening K.L., Edema and brain trauma, Neuroscience, 2004, 129, 1021–1029 http://dx.doi.org/10.1016/j.neuroscience.2004.06.046CrossrefGoogle Scholar
[23] Wagner S., Nagel S., Kluge B., Schwab S., Heiland S., et al., Topographically graded postischemic presence of metalloproteinases is inhibited by hypothermia, Brain Res., 2003, 984, 63–75 http://dx.doi.org/10.1016/S0006-8993(03)03088-9CrossrefGoogle Scholar
[24] Montaner J., Alvarez-Sabin J., Barbera G., Angles A., Molina C., et al., Correlation between the expression of proinflammatory cytokines and matrix metalloproteinases in the acute phase of an ischemic stroke, Rev. Neurol., 33, 115–118 Google Scholar
Comments (0)